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Since the 1950s, corticosteroids (steroids) have been helping those with ulcerative colitis (UC) put the disease in remission. There is a lot of uncertainty, however, about whether steroid use actually works as well as the current clinical trials suggest. Now, a team of researchers at the Fred Hutchinson Cancer Research Center (FHRC) in Seattle is studying a different kind of treatment for UC: what they call "dose-response" therapy. Instead of using steroids as a last resort and trying to keep the inflammatory response in check in one or several patients, the FHRC team is encouraging doctors to try to stop UC altogether, often with the help of "placebo" medicines to mask symptoms like diarrhea and weight loss. Although steroidal therapy can improve remission rates and even cure some patients of their illness, there are some potential risks. Because the FHRC team hopes its treatments have long-lasting effects, it is starting with a control group of people who receive steroid-based therapy as a daily treatment. Researchers hope to identify better treatments to prevent relapse from drug treatment. According to the NIH, a small but growing number of patients with UC have reported improvements in their condition after starting chemotherapy, surgery, and radiation therapy. But because these treatments can be very harsh and sometimes lead certain patients to relapse, they represent a small fraction of patients who are likely to benefit from these treatments. Now, the researchers want to study the long-term benefits of their treatments to determine exactly how effective these treatments can be. The team plans to look at two different classes of anti-inflammatory drugs called natalistein, which they have been developing before, and the new anti-inflammatory medicine rotenone, which will be tested shortly. The researchers are using a new experimental treatment called immunotherapeutic antibody (inA). This type of antibody binds to proteins inside the UC, so it is designed to target one or several specific inflammatory cells instead of suppressing the entire immune response. In a paper published in March in the journal Biomarkers in Immunotherapy, the researchers describe how they hope to study if natalistein therapy can delay the start of inflammation in people with UC before they start taking other side effects, like headaches. If it works, it may be worth treating many more patients before the therapy does damage, says lead study investigator Dr. William Schmoll, chief of the Cancer Medicine Branch at the FHRC. While natalistein was first developed for a cancer condition called immunoreactive T-cell lymphoma, Dr. Schmoll says Related Article:

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